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Keywords: T cells
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Pathobiology (1996) 64 (1): 27–31.
Published Online: 08 October 2008
... is due in part to activation of T lymphocytes. DGLA suppresses T cell activation and production of interleukin-2 (IL-2). The protooncogenes c-myc and c-fos are early response genes which are critical to regulation of T cell proliferation. We therefore examined the effects of γ-linolenic acid and other...
Journal Articles
Experimental Cell Biology (1985) 53 (3): 150–162.
Published Online: 07 October 2008
... concentrations (SMLC). Supernates of parental T cells reacting with F 1 spleen cells also produce this factor. The product ameliorates SMLC proliferative responses specifically since identical antigenic specificities have to be recognized in both SEPIR formation and in test SMLC. Further evidence for SEPIR being...
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Experimental Cell Biology (1985) 53 (3): 138–149.
Published Online: 07 October 2008
... leukocyte cultures (MLC) set up in suboptimal conditions. The factor appears to be generated by parental strain T but not B lymphocytes, is active at low concentration and acts in a pulse-like fashion. Its formation is triggered by unstimulated T cells reacting with H-2 antigens; no cytokine activity of IL...
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Experimental Cell Biology (1979) 47 (2): 107–118.
Published Online: 03 October 2008
...H. Ramseier Alloantisera could be induced in parental strain recipients either by means of alloantigen (skin allografting) or by injecting anti-idiotypic (anti-T cell receptor) sera. The resulting sera displayed activity against H-2 antigens; they also contained soluble idiotypic structures...
Journal Articles
Experimental Cell Biology (1977) 45 (1-2): 101–117.
Published Online: 03 October 2008
...H. Ramseier; J. Lindenmann The product of antigenic recognition (PAR) is an antigen-antibody complex with granulotactic activity. The following results support his conclusion. PAR generated by cell-bound or shed T-cell RS or the recognizing structure of T alloantisera and alloantigen can...
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Experimental Cell Biology (1977) 45 (1-2): 89–100.
Published Online: 03 October 2008
... and J. L indenmann Division of Experimental Microbiology, Institute for Medical Microbiology, University of Zurich, Zurich Key Words: Recognition . Receptors . T cells T alloantisera . Anti-receptor sera . Anti-alloantisera Abstract. Cell-bound or shed T-lymphocyte receptors for alloantigens...
Journal Articles
Experimental Cell Biology (1977) 45 (1-2): 60–88.
Published Online: 03 October 2008
...H. Ramseier; J. Lindenmann A product of antigenic recognition (PAR) was produced whenever receptors for alloantigens from T lymphocytes or a principle present in T-cell dependent alloantisera interacted with alloantigen. With two forms of the PAR assay (direct and indirect) the mechanisms...
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Journal Articles
Pathobiology (2003) 70 (3): 121–130.
Published Online: 12 February 2003
... or services advertised or of their effectiveness, quality or safety. The publisher and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Classification T cells Crohn’s...
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Pathobiology (1998) 66 (6): 302–305.
Published Online: 16 October 1998
... was expected but failed to inhibit the proliferative response to silicon dioxide. Further testing with monoclonal antibodies to HLA-DP, -DQ, and a second -DR with specificity for the NS1 region of the MHC class II genome, all markedly inhibited proliferation of T cells despite otherwise adequate stimulation...
Journal Articles
Pathobiology (1998) 66 (3-4): 151–157.
Published Online: 23 July 1998
... demonstrated a more pronounced loss of CD4+ T cells in the intestinal mucosa compared to the peripheral blood in humans infected with HIV. In parallel, activated CD8+ T cells in the lamina propria are increased in this compartment. In SIV-infected nonhuman primates a very early loss of CD4+ T cells...