Skip Nav Destination
Close Modal
Update search
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
Filter
All
- All
- Title
- Author
- Author Affiliations
- Full Text
- Abstract
- Keyword
- DOI
- ISBN
- EISBN
- ISSN
- EISSN
- Issue
- Volume
- References
NARROW
Format
Subjects
Journal
Article Type
Date
Availability
1-4 of 4
Keywords: Tumor necrosis factor-α
Close
Follow your search
Access your saved searches in your account
Would you like to receive an alert when new items match your search?
Sort by
Journal Articles
Dominant Negative p38 Mitogen-Activated Protein Kinase Expression Inhibits NF-ĸB Activation in AR42J Cells
Available to PurchaseSubject Area:
Gastroenterology
Journal:
Pancreatology
Pancreatology (2010) 10 (2-3): 119–128.
Published Online: 07 May 2010
... incorporating an NF-ĸB-responsive luciferase reporter, with and without adenoviral transduction of DNp38, were stimulated with cholecystokinin (CCK) or tumor necrosis factor-α (TNF-α) prior to measuring NF-ĸB activation. Results: CCK- or TNF-α-stimulated NF-ĸB-dependent gene transcription (luciferase assay...
Journal Articles
Chronic Pancreatitis: Evolving Paradigms
Available to PurchaseSubject Area:
Gastroenterology
Journal:
Pancreatology
Pancreatology (2006) 6 (5): 440–449.
Published Online: 25 October 2006
... and pancreatic fibrosis is now better understood. Molecular mediators shown to regulate the pathogenesis include transforming growth factor-β, platelet-derived growth factor, and proinflammatory cytokines such as interleukin (IL)-1, IL-6 and tumor necrosis factor-α. Besides these, the roles of cyclooxygenase...
Journal Articles
Pro−Inflammatory Cytokine−Induced Matrix Metalloproteinase−1 (MMP−1) Secretion in Human Pancreatic Periacinar Myofibroblasts (Kazuhito Tasaki et al., Otsu)
Available to PurchaseSubject Area:
Gastroenterology
Kazuhito Tasaki, Yutaka Shintani, Takao Saotome, Akira Andoh, Yoshihide Fujiyama, Shigenari Hozawa, Tadao Bamba
Journal:
Pancreatology
Pancreatology (2003) 3 (5): 414–421.
Published Online: 29 October 2003
... in chronic pancreatitis via MMP-1 secretion. 3 12 2002 8 07 2003 29 10 2003 Interleukin-1β Matrix metalloproteinases MEK Mitogen-activated protein kinase Tumor necrosis factor-α Original Paper Pancreatology 2003;3:414 421 DOI: 10.1159/000073889 Received: December 3, 2002 Revised...
Journal Articles
Leptin Protects the Pancreas from Damage Induced by Caerulein Overstimulation by Modulating Cytokine Production
Available to PurchaseSubject Area:
Gastroenterology
Jolanta Jaworek, Joanna Bonior, Piotr Pierzchalski, Romana Tomaszewska, Jerzy Stachura, Ryszard Sendur, Anna Leja, Bożena Jachimczak, Peter C. Konturek, Władysław Bielański, Wiesław Pawlik, Stanisław J. Konturek
Journal:
Pancreatology
Pancreatology (2002) 2 (2): 89–99.
Published Online: 24 April 2002
... to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Leptin Caerulein-induced pancreatitis ob gene expression Tumor necrosis factor-α Interleukin-4 Pancreatic acini Pancreatic blood flow Original Paper Pancreatology...