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Keywords: Glucocorticoid receptor
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Neuroimmunomodulation (2014) 22 (1-2): 6–19.
Published Online: 12 September 2014
... to in the content or advertisements. Stress system Stress response Glucocorticoids Glucocorticoid receptor Hypothalamic-pituitary-adrenal axis All living organisms must respond to many unforeseen extrinsic or intrinsic stressful stimuli, the stressors, which constantly challenge our complex...
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Neuroimmunomodulation (2014) 22 (1-2): 40–45.
Published Online: 12 September 2014
... or safety. The publisher and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Rheumatoid arthritis Glucocorticoid receptor 11β-Hydroxysteroid dehydrogenases...
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Neuroimmunomodulation (2014) 22 (1-2): 20–32.
Published Online: 12 September 2014
... transduction by their steroid receptor, the glucocorticoid receptor (GR). The mechanisms modulating the pro-inflammatory effects of glucocorticoids are not well understood. In this review, we discuss recent findings that provide insights into the mechanism by which GR signaling can play a dual role...
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Neuroimmunomodulation (2011) 18 (4): 199–211.
Published Online: 19 February 2011
...-6 and tumor necrosis factor-α were determined by enzyme-linked immunosorbent assays. Immune function was assessed by in vitro natural killer cell cytotoxicity (NKCC). Lymphocyte counts, immunophenotype and intracellular glucocorticoid receptor expression in various lymphocyte subsets were determined...
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Neuroimmunomodulation (2008) 15 (1): 19–28.
Published Online: 29 July 2008
... transporter 11 -Hydroxysteroid dehydrogenase Glucocorticoid receptor Cytokines Inflammation Autoimmune disease Abstract Rheumatoid arthritis (RA) is characterized by chronic inflam- mation of the synovial membrane, leading to joint destruc- tion. Many autoimmune diseases and disease states...
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Neuroimmunomodulation (1995) 2 (6): 339–346.
Published Online: 12 July 1996
... corticosteroid receptor antagonists, RU28362 and RU38486, for the displacement of labeled dexamethasone revealed that binding was mainly of type-II glucocorticoids receptors. In lymphocytes obtained from mesenteric lymph nodes, binding was significantly higher than in cells obtained from spleen and thymus. Long...
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