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Keywords: Haloperidol
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Journal Articles
Journal Articles
Neuroendocrinology (1992) 55 (5): 519–528.
Published Online: 07 April 2008
... haloperidol resulted in significant increases in plasma prolactin levels. Mecamylamine pretreatment prevented the prolactin response to nicotine only. N-altrexone blocked the stimulation of prolactin release by morphine and by nicotine. Methysergide inhibited the effects of 8-OH-DPAT, morphine and nicotine...
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Neuroendocrinology (1987) 45 (5): 333–337.
Published Online: 02 April 2008
... of response of tuberoinfundibular DA neurons to stress is related to the reduced basal activity of these neurons in the aged rat, the response of tuberoinfundibular DA neurons to 30 min of restraint was determined in rats that were pretreated with haloperidol. Haloperidol increased serum prolactin levels...
Journal Articles
Neuroendocrinology (1987) 45 (3): 227–232.
Published Online: 02 April 2008
... in the median eminence of the aged (26 months) rat was reduced to 50–60% of that in the young (3 months) rat. The acute systemic administration of haloperidol, a DA antagonist which increases serum concentrations of prolactin or intracerebroventricular infusions of prolactin increased the rate of dopa...
Journal Articles
Neuroendocrinology (1986) 44 (4): 462–469.
Published Online: 01 April 2008
... but not kallikrein substrates. This study examined whether any of the pituitary proteases are regulated by dopaminergic mechanisms. Ovariectomized female rats were treated for 5–10 days with reserpine (a catecholamine depleting agent), haloperidol (a dopamine receptor blocker) or bromocriptine (a dopamine receptor...
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Journal Articles
Neuroendocrinology (1986) 43 (3): 368–376.
Published Online: 01 April 2008
...–6 days with various doses of the following drugs, alone or in combination: haloperidol (a dopamine receptor blocker), reserpine (a catecholamine depleting agent), and bromocriptine (a dopamine receptor agonist). Neurointermediate lobe () homogenates were prepared. Following activation of latent...
Journal Articles
Neuroendocrinology (1986) 42 (3): 191–196.
Published Online: 01 April 2008
... an interaction exists between these two reciprocal mechanisms regulating β-END-LI in the rat. The intraperitoneal (i.p.) administration of 5 mg/kg quipazine, a serotonin receptor agonist, or 2.5 mg/kg haloperidol, a dopamine receptor antagonist, each elevated circulating levels of β-END-LI 5-fold over control...
Journal Articles
Neuroendocrinology (1985) 40 (2): 185–187.
Published Online: 28 March 2008
...Luciano Debeljuk; Ignacio Torres-Aleman; Andrew V. Schally We investigated the effect of blocking endogenous LH-RH, by injection of anti-LH-RH sera, on serum prolactin levels previously elevated by treatment with haloperidol for 5 days. An acute intravenous injection of rabbit anti-LH-RH serum...
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Neuroendocrinology (1981) 33 (2): 97–100.
Published Online: 26 March 2008
...J.M. Jacobi; H.M. Lloyd Pituitary DNA synthesis and serum and pituitary concentrations of prolactin were measured in male rats given the dopamine antagonist haloperidol or pimozide, and the dopamine agonist bromocriptine. Increased DNA synthesis was observed 24 h after a single injection...
Journal Articles
Journal Articles
Neuroendocrinology (1982) 34 (5): 369–373.
Published Online: 26 March 2008
... haloperidol, or by serotonergic receptor activation produced by fluoxetine, a serotonergic reuptake inhibitor plus 5-hydroxytryptophan, a serotonin precursor. Inhibition of prolactin release by benzodiazepine was dose related, and inhibition was still evident after repeated diazepam injection. The potency...
Journal Articles
Neuroendocrinology (1980) 30 (2): 65–69.
Published Online: 26 March 2008
... agent haloperidol (HAL). DOM proved to be a potent PRL releaser since striking and long-lasting (120 min) rises in plasma PRL were elicited by 50 and 100 μ g/kg of the compound. Comparison with HAL showed that DOM was 5 times as potent as HAL as a PRL releaser (ED 250 = 0.084 vs. 0.42 mg/kg...
Journal Articles