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Keywords: Reactive oxygen species
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Journal Articles
Subject Area:
Nephron Clinical Practice (2014) 127 (1-4): 124–128.
Published Online: 24 September 2014
... and subcellular constituents as being central to the etiology of AKI. Injury is caused by inflammatory activation involving endothelial leucocyte interactions in combination with dysregulation of the homeostatis between oxygen, nitric oxide, and reactive oxygen species. Effective therapies expected to resolve AKI...