1-1 of 1
Keywords: Reactive oxygen species
Close
Follow your search
Access your saved searches in your account

Would you like to receive an alert when new items match your search?
Close Modal
Sort by
Journal Articles
Subject Area:
Nephrology
Nephron Clinical Practice (2014) 127 (1-4): 124–128.
Published Online: 24 September 2014
... and subcellular constituents as being central to the etiology of AKI. Injury is caused by inflammatory activation involving endothelial leucocyte interactions in combination with dysregulation of the homeostatis between oxygen, nitric oxide, and reactive oxygen species. Effective therapies expected to resolve AKI...