... of the aorto-caval fistula (ACF). Renal blood flow (RBF) responses were determined to renal arterial administration of ANG II, native 11,12-EET, an analog of 14,15-EETs (EET-A), norepinephrine (NE), acetylcholine (Ach) and bradykinin (Bk) in healthy (i.e., sham-operated) TGR and ACF TGR (5 weeks after ACF...

... by an ultrasonic transient-time flow probe maximum RBF responses to renal artery infusion of angiotensin II (ANG II), norepinephrine (NE) and acetylcholine (Ach) in healthy male rats and animals with compensated and decompensated CHF. CHF was induced by volume overload achieved by the creation of the aorto-caval...

...Gibson Oriji; John R. Gill, Jr.; John C.H. Yun; Harry Keiser Renal arterial infusion of acetylcholine (ACh; 40 µg/min) in control dogs produced a sustained rise in urinary sodium excretion (U Na V) and in renal plasma flow (RPF). When prostaglandin (PG) synthesis was inhibited with indomethacin (5...

... (an index of EDRF production) was determined by radioimmunoassay under basal conditions and after acetylcholine (10 -5 M ), bradykinin (10 -5 M ) and SIN-1 (10 -5 M ) stimulation. Under basal and stimulated conditions, the cGMP concentrations were highest in the midinner medulla and decreased progressively...

...Ozra Dehkordi; John C.H. Yun; Sai-Sun Ho; John R. Gill, Jr.; Harry Keiser Renal arterial infusion of acetylcholine (ACh) in the dog normally produces a sustained rise in sodium excretion (U Na V) and in renal plasma flow (RPF). When prostaglandin (PG) synthesis is inhibited, ACh induces only...

...–dependent (acetylcholine–induced) and/or endothelium–independent (sodium nitroprusside–induced) vasorelaxation and to evaluate basal NO release by the infusion of N G –nitro–L–arginine methyl ester (L–NAME) in an isolated and perfused rat kidney model. Methods: To investigate this hypothesis, we have...