... and the editor(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Taurine Cysteine sulfinic acid decarboxylase GABA L-Glutamate decarboxylase Mn^2+ Journal cf Biomedicai Science...

... preparations in the presence of protease inhibitors. Furthermore, full-length GAD could be converted to the truncated form by endogenous proteases, suggesting that the conversion of full-length to truncated GAD mediated by endogenous protease may represent an important mechanism in the regulation of GABA...

... decarboxylase (GAD65) by molecular oxygen is likely to result in incomplete filling of synaptic vesicles with γ-aminobutyric acid (GABA) and may be a contributing factor in the genesis of oxygen-induced seizures. Under anaerobic conditions, nitric oxide inhibits both GAD65 and GAD67 with comparable potency...

... responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. L -Glutamate GABA Alcoholism Glutamate receptors GABA receptors Decarboxylase Review J Biomed Sci 2001;8:7 19 Role of Glutamatergic...

...Mina Yarom; Xiao Wen Tang; Elliott Wu; Robert G. Carlson; David Vander Velde; Xi-Hsuan Lee; Jang-Yen Wu Previously we have reported the presence of endogenous ligands that are involved in the regulation of the binding of muscimol to the GABA binding site of the GABA A receptors. Here, we report...