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Keywords: Protein kinase C
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Journal Articles
Journal Articles
Dev Neurosci (1995) 17 (5-6): 278–284.
Published Online: 28 May 2010
..., researchers have begun to investigate the mechanisms that regulate gliogenesis, glial cell differentiation and myelinogenesis. The progress made so far strongly implicates protein kinase C (PKC)- and protein kinase A (PKA)-mediated signaling in glial cell proliferation and differentiation. These pathways...
Journal Articles
Dev Neurosci (1995) 17 (5-6): 267–277.
Published Online: 28 May 2010
..., researchers have begun to investigate the mechanisms that regulate gliogenesis, glial cell differentiation and myelinogenesis. The progress made so far strongly implicates protein kinase C (PKC)- and protein kinase A (PKA)-mediated signaling in glial cell proliferation and differentiation. These pathways...
Journal Articles
Dev Neurosci (1995) 17 (4): 256–263.
Published Online: 28 May 2010
...Narayan R. Bhat; Peisheng Zhang; Aruna N. Bhat Myristoylated alanine-rich C-kinase substrate (MARCKS), a major substrate of activated protein kinase C (PKC), is thought to be involved in PKC-mediated signal transduction events. In the present study, we have examined the expression of MARCKS...
Journal Articles
Dev Neurosci (2010) 32 (1): 33–46.
Published Online: 03 December 2009
... markers, intracellular cyclic adenosine monophosphate (cAMP) levels, protein kinase C (PKC) signal transduction to extracellular signal-regulated kinase 1/2 (ERK-1/2) and cellular morphology were investigated in relation to RKIP levels. RKIP + cells showed accelerated neurite outgrowth, formation...
Journal Articles
Journal Articles
Dev Neurosci (2005) 26 (5-6): 371–379.
Published Online: 20 April 2005
...Flavio R. Zolessi; Cristina Arruti Myristoylated alanine-rich kinase C substrate (MARCKS), an actin-binding protein, is involved in several signal transduction pathways. It is susceptible to be phosphorylated by protein kinases as protein kinase C and some proline-directed kinases...
Journal Articles
Journal Articles
Journal Articles
Dev Neurosci (1989) 11 (2): 118–131.
Published Online: 13 June 1989
... in OLGs can be inhibited by two distinct second messengers, cAMP acting through protein kinase A and diacylglycerol acting through protein kinase C, the effects of which perhaps converge at the level of a common phosphorylated enzyme or regulatory protein. Both cAMP and diacylglycerol have been implicated...