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Keywords: IPEC-J2
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Journal Articles

Crosstalk Between Nuclear Glucose-Regulated Protein 78 and Tumor Protein 53 Contributes to the Lipopolysaccharide Aggravated Apoptosis of Endoplasmic Reticulum Stress-Responsive Porcine Intestinal Epithelial Cells

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Qian Jiang, Gang Liu, Jiashun Chen, Kang Yao, Yulong Yin
Journal: Cellular Physiology and Biochemistry
Cellular Physiology and Biochemistry (2018) 48 (6): 2441–2455.
https://doi.org/10.1159/000492682
Published Online: 17 August 2018
... reticulum stress (ERS) and the subsequent apoptosis of intestinal epithelial cells. Therefore It is important to explore whether LPSs aggravate ERS-mediated apoptosis of intestinal epithelial cells. Methods: ERS and inflammation models were established in porcine cell line J2 (IPEC-J2) and the cells were...
View article titled, Crosstalk Between Nuclear Glucose-Regulated Protein 78 and Tumor Protein 53 Contributes to the Lipopolysaccharide Aggravated Apoptosis of Endoplasmic Reticulum Stress-Responsive Porcine Intestinal Epithelial Cells
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Journal Articles

Clostridium Tyrobutyricum Protect Intestinal Barrier Function from LPS-Induced Apoptosis via P38/JNK Signaling Pathway in IPEC-J2 Cells

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Zhiping Xiao, Lujie Liu, Wenjing Tao, Xun Pei, Geng Wang, Minqi Wang
Journal: Cellular Physiology and Biochemistry
Cellular Physiology and Biochemistry (2018) 46 (5): 1779–1792.
https://doi.org/10.1159/000489364
Published Online: 25 April 2018
... ) on intestinal barrier dysfunction induced by lipopolysaccharide (LPS) and the underlying mechanisms involved in intestinal barrier protection. Methods: A Transwell model of IPEC-J2 cells was used to imitate the intestinal barrier. Fluorescence microscopy and flow cytometry were used to evaluate apoptosis. Real...
View article titled, Clostridium Tyrobutyricum Protect Intestinal Barrier Function from LPS-Induced Apoptosis via P38&#x2F;JNK Signaling Pathway in <span class="search-highlight">IPEC</span>-<span class="search-highlight">J2</span> Cells
Open the PDF for Clostridium Tyrobutyricum Protect Intestinal Barrier Function from LPS-Induced Apoptosis via P38/JNK Signaling Pathway in <span class="search-highlight">IPEC</span>-<span class="search-highlight">J2</span> Cells in another window
Journal Articles

AMP010014A09 in Sus Scrofa Encodes an Analog of G Protein-Coupled Receptor 109A, Which Mediates the Anti-Inflammatory Effects of Beta-Hydroxybutyric Acid

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Guangxin Chen, Shoupeng Fu, Wenqian Feng, Bingxu Huang, Shiyao Xu, Wei Wang, Juxiong Liu
Journal: Cellular Physiology and Biochemistry
Cellular Physiology and Biochemistry (2017) 42 (4): 1420–1430.
https://doi.org/10.1159/000479206
Published Online: 17 July 2017
... after pretreating with or without β-hydroxybutyric acid (BHBA), and the levels of pro-inflammatory cytokines and inflammatory proteins were assessed. cAMP levels induced by Forskolin in swine testicular (ST) and IPEC-J2 cells were detected with or without BHBA treatment and following silencing or stable...
View article titled, AMP010014A09 in Sus Scrofa Encodes an Analog of G Protein-Coupled Receptor 109A, Which Mediates the Anti-Inflammatory Effects of Beta-Hydroxybutyric Acid
Open the PDF for AMP010014A09 in Sus Scrofa Encodes an Analog of G Protein-Coupled Receptor 109A, Which Mediates the Anti-Inflammatory Effects of Beta-Hydroxybutyric Acid in another window