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Keywords: Hypertrophy
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Cellular Physiology and Biochemistry (2018) 48 (1): 293–303.
Published Online: 17 July 2018
...) and is a high-morbidity problem in the elderly. LSS is commonly caused by hypertrophy of the ligamentum flavum (HLF). Previous studies showed that fibrosis of the ligamentum flavum (LF) largely contributed to HLF. However, the underlying pathomechanism remains unclear. Insulin-like growth factor-1 (IGF-1...
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Cellular Physiology and Biochemistry (2018) 47 (1): 140–150.
Published Online: 10 May 2018
... regulator of the gp130 receptor to inhibit cardiac hypertrophy. However, the role of SOCS3 in deoxycorticosterone-acetate (DOCA)-salt-induced cardiac remodeling remains unclear. Methods: Cardiac-specific SOCS3 knockout (SOCS3cKO) and wild-type (WT) C57BL/6J mice were subjected to uninephrectomy and DOCA...
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Cellular Physiology and Biochemistry (2017) 43 (4): 1449–1459.
Published Online: 11 October 2017
... involved in glucose use. In addition, RA increased JNK 1/2 expression, without changes in TGF-β, PI3K, AKT, NFκB, S6K, and ERK. Conclusion: In normal rats, RA induces cardiac hypertrophy in a dose-dependent manner. The non-participation of the PI3K/Akt pathway, associated with the participation of the JNK...
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Cellular Physiology and Biochemistry (2017) 41 (5): 1830–1837.
Published Online: 03 April 2017
...Shutong Shen; Huimin Jiang; Yihua Bei; Junjie Xiao; Xinli Li Cardiac remodeling occurs after stress to the heart, manifested as pathological processes, including hypertrophy and apoptosis of cardiomyocytes, dysfunction of vascular endothelial cells and vascular smooth muscle cells as well...
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Cellular Physiology and Biochemistry (2017) 41 (3): 849–864.
Published Online: 16 February 2017
... baicalin plays a role in cardiac hypertrophy remains unknown. Here we investigated the protective effects of baicalin on cardiac hypertrophy induced by pressure overload and explored the potential mechanisms involved. Methods : C57BL/6J-mice were treated with baicalin or vehicle following transverse aortic...
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Cellular Physiology and Biochemistry (2016) 38 (5): 1743–1751.
Published Online: 09 May 2016
... and hypertrophy in response to hypertensive or ischemic stress. However, little is known about whether QL could modulate cardiac hypertrophy in vitro , and (if so) whether it is through modulation of specific hypertrophy-related microRNA. Methods: The primary neonatal rat ventricular cardiomyocytes were isolated...
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Cellular Physiology and Biochemistry (2015) 36 (5): 1688–1698.
Published Online: 13 July 2015
... contribute to the etiology of cardiac hypertrophy. Methods: Adult male C57BL/6J mice were subjected to pressure overload by using transverse aortic constriction (TAC), and then received genipin (a UCP2 selective inhibitor; 25 mg/kg/d, ip) or vehicle for three weeks prior to histologic assessment...
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Cellular Physiology and Biochemistry (2014) 34 (4): 1241–1259.
Published Online: 29 September 2014
...Caroline Brun; Luce Périé; Fabienne Baraige; Barbara Vernus; Anne Bonnieu; Véronique Blanquet Background/Aims: Overexpression of Gasp-1 , an inhibitor of myostatin, leads to a hypermuscular phenotype due to hypertrophy rather than hyperplasia in mice. However to date, the cellular and molecular...
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Cellular Physiology and Biochemistry (2011) 26 (6): 879–886.
Published Online: 04 January 2011
... in diseases such as obesity and diabetes, with the onset of diabetic nephropathy associated with proximal tubule hypertrophy. Recent research has identified a role for CB1 in apoptosis in human proximal tubular (HK2) cells, however the role of the other receptors has not been investigated. We investigated...
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Cellular Physiology and Biochemistry (2009) 24 (1-2): 1–10.
Published Online: 01 July 2009
...(s) disclaim responsibility for any injury to persons or property resulting from any ideas, methods, instructions or products referred to in the content or advertisements. Dexamethasone Hypertension Hypertrophy Myocardium Oxidative Stress Original Paper Cellular Physiology Cell Physiol...
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Cellular Physiology and Biochemistry (2009) 23 (4-6): 327–334.
Published Online: 06 May 2009
... expression in the left ventricle (LV) was followed by LV hypertrophy. Methods: Using IL-6 deficient mice (IL-6 -/- ), we studied the role of IL-6 in a model of norepinephrine (NE)-induced LV hypertrophy. Results: In wild type (WT) mice, IL-6 mRNA expression and its concentration in the serum were elevated...
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