Hyposexuality after stroke has been frequently observed, but hypersexuality as a sequela of stroke has been less commonly documented. Damage to limbic structures, especially in the temporal lobes, has been thought to play a crucial role in this clinical syndrome. The possible importance of the basal ganglia in the production of hypersexuality has been infrequently recognized despite numerous connections with the limbic system. We report two patients who developed intense hypersexuality following strokes (one ischemic, the other hemorrhagic) in the region of the basal ganglia. This expands the spectrum of cerebral lesions implicated in hypersexuality, and warrants systematic questioning as to patients'' sexual function after basal ganglia strokes, since such information may not be spontaneously offered.

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