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syndrome

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Book Chapter
Book Chapter
Series: Frontiers of Hormone Research
Volume: 49
Published: 26 April 2018
10.1159/000486002
EISBN: 978-3-318-06335-6
... Abstract Cushing's syndrome (CS), including visceral obesity, dyslipidemia, hypertension and diabetes among its many manifestations, is “a model” of metabolic syndrome. Glucocorticoid (GC) excess, through a combination of effects on liver, muscle, adipose tissue and pancreas, increases...
Book Chapter
Series: Frontiers of Hormone Research
Volume: 49
Published: 26 April 2018
10.1159/000485995
EISBN: 978-3-318-06335-6
... Abstract Both prevalence and incidence of the metabolic syndrome is very high in women with polycystic ovary syndrome (PCOS). Obesity and visceral fat enlargement play a dominant role in determining the final phenotype of PCOS. Androgen excess and insulin resistance may be responsible...
Book Chapter
Series: Frontiers of Neurology and Neuroscience
Volume: 41
Published: 06 December 2017
10.1159/000475701
EISBN: 978-3-318-05859-8
... Abstract Different eponyms such as “Wood syndrome,” Meige syndrome, “Brueghel syndrome,” “Blepharospasm plus syndrome” have been used to describe segmental craniocervical dystonias. These facial and/or oromandibular movement disorders are characterized by muscle contractions and spasms...
Book Chapter
Series: Frontiers of Neurology and Neuroscience
Volume: 42
Published: 06 December 2017
10.1159/000475685
EISBN: 978-3-318-06089-8
..., and somatic. We present here 2 main delusional disorders, the De Clérambault syndrome and the Othello syndrome, and another closely related to the previous ones - Folie à deux. In the De Clérambault syndrome, the main delusional theme is erotomanic type, related to passional delirium where the patient has...
Book Chapter
Series: Frontiers of Hormone Research
Volume: 40
Published: 19 October 2012
10.1159/000341840
EISBN: 978-3-318-02239-1
... Abstract The complex mechanisms linking fat excess to metabolic syndrome are not well understood, but several experimental studies have shown that altered production of adipokines plays a main role in development and progression of this disorder. In particular, reduced secretion of adiponectin...
Book Chapter
Series: Endocrine Development
Volume: 13
Published: 05 June 2008
10.1159/000134829
EISBN: 978-3-8055-8581-1
... Abstract Endogenous Cushing syndrome (CS) is caused by excess adrenal glucocorticoid secretion that is adrenocorticotropin(ACTH)-dependent or independent; ACTH-independent adrenocortical causes of CS account forup to 20% of CS in adults, and 15% in children over age 7 years. In younger children...
Book Chapter
Series: Advances in Cardiology
Volume: 45
Published: 14 February 2008
10.1159/000115191
EISBN: 978-3-8055-8428-9
... Abstract The reported incidence of metabolic syndrome among patients with an acute coronarysyndrome varies between 29 and 46%. The standard fasting cut-off levels for glucoseand blood pressure cannot be applied on admission in patients with acute coronary syndromeand therefore modified criteria...
Book Chapter
Series: Developments in Ophthalmology
Volume: 37
Published: 18 June 2003
10.1159/000072043
EISBN: 978-3-318-00971-2
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Volume: 31
Published: 31 December 1979
10.1159/000287325
EISBN: 978-3-318-06164-2
... individuals. Moreover the author differentiates between the amygdaloid nucleus and dehumanization syndromes, through psychoanalytical or psychiatric studies, and stresses the importance of the molding periods in relation to social factors (frustration-aggression-injustice). Amygdalotomy of the hippocampus...
Book Chapter
Published: 17 December 2024
10.1159/000538204
EISBN: 978-3-318-07273-0
... surrounding and supporting blood vessels (connective tissue disorders) or in the blood vessels themselves, as in hereditary hemorrhagic telangiectasia (HHT). While connective tissue disorders include several very rare conditions such as Marfan syndrome, Loeys-Dietz syndrome, and homocystinuria, the most...
Book Chapter
Series: World Review of Nutrition and Dietetics
Volume: 127
Published: 04 March 2024
10.1159/000534807
EISBN: 978-3-318-07341-6