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gout

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Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
10.1159/000484281
EISBN: 978-3-318-06251-9
... Abstract As a disease of kings, and the king of diseases, gout is one of the oldest joint diseases known to humans. First described as far back as 2640 B.C., gout is still the most common form of inflammatory arthritis haunting humans in the 21st century. The disease is caused by the chronic...
Book Chapter
Published: 17 August 2012
10.1159/000336433
EISBN: 978-3-8055-9930-6
... Abstract Gout is a common form of arthritis caused by the deposition of proinflammatory monosodium urate crystals in the setting of chronic hyperuricemia (serum urate concentration >7 mg/dl, or 0.42 m<smlcap>m</smlcap>). The risks of becoming hyperuricemic and developing gout...
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Book Chapter
Series: Contributions to Nephrology
Volume: 147
Published: 07 December 2004
10.1159/000082538
EISBN: 978-3-318-01169-2
... Abstract Gout is among the most common causes of acute monoarticular arthritis. It is characterizedby depositions of monosodium urate crystals in a joint or in soft tissues, causing anacute inflammatory response. Gout typically occurs in middle age and more commonly inmen. The four phases...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
EISBN: 978-3-318-06251-9
... BJ, Choi HK: Prevalence of gout and hyperuricemia in the US general population: the National Health and Nutrition Examination Survey 2007-2008. Arthritis Rheum 2011;63:3136-3141. 4. Harris CM, Lloyd DC, Lewis J: The prevalence and prophylaxis of gout in England. J Clin Epidemiol 1995;48:1153-1158...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
EISBN: 978-3-318-06251-9
...:182-192. 6. Borghi C, Rosei EA, Bardin T, Dawson J, Dominiczak A, Kielstein JT, et al: Serum uric acid and the risk of cardiovascular and renal disease. J Hypertens 2015;33:1729-1741. 7. Chen JH, Yeh WT, Chuang SY, Wu YY, Pan WH: Gender-specific risk factors for incident gout: a prospective...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
10.1159/000484288
EISBN: 978-3-318-06251-9
... intervention trials that linked consumed quantities of individual dietary components to changes in SUA [ 13 ]. Most of the recommendations are taken from studies of patients with gout and are useful for treating other life-threatening comorbidities of hyperuricemic patients, including obesity, coronary artery...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
EISBN: 978-3-318-06251-9
... identified urate transporter influencing serum urate concentration, urate excretion and gout. Nat Genet 2008;40:437-442. 12. Phay JE, Hussain HB, Moley JF: Cloning and expression analysis of a novel member of the facilitative glucose transporter family, SLC2A9 (GLUT9). Genomics 2000;66:217-220. 13...
Book Chapter
Published: 17 August 2012
EISBN: 978-3-8055-9930-6
...References References 1. Bhole V, de Vera M, Rahman MM, Krishnan E, Choi H: Epidemiology of gout in women: fifty-two-year followup of a prospective cohort. Arthritis Rheum 2010;62:1069-1076 2. Lawrence RC, Felson DT, Helmick CG, Arnold LM, Choi H, Deyo RA, et al: Estimates...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
10.1159/000484275
EISBN: 978-3-318-06251-9
... diseases such as lithiasis, gout nephropathy, and preeclampsia. In the last 30 years, it has been shown that reducing HyU with low protein and low purine diets in addition to allopurinol creates physiopathological conditions that produce a slight increase in the glomerular filtration rate (GFR). In recent...
Book Chapter
Series: Developments in Ophthalmology
Volume: 48
Published: 20 April 2011
10.1159/000324078
EISBN: 978-3-8055-9721-0
... misdiagnosed. The differential diagnosis of the SCD patient includes other diseases with crystalline deposits such as cystinosis, tyrosinemia, Bietti crystalline dystrophy, hyperuricemia/gout, multiple myeloma, monoclonal gammopathy, infectious crystalline keratopathy, and Dieffenbachia keratitis. Depositions...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
10.1159/000484286
EISBN: 978-3-318-06251-9
... URAT1 was a breakthrough in our understanding of urate handling by the nephron, and it is the transporter that is defective in familial hypouricemic hyperuricosuria. To date, 11 different loss-of function mutations have been identified [ 21 ]. Most patients with primary gout hyperuricemia have...
Book Chapter
Series: Contributions to Nephrology
Volume: 147
Published: 07 December 2004
10.1159/000082548
EISBN: 978-3-318-01169-2
... homeostasis. Although there isonly indirect evidence (derived from obstetric eclampsia and experimental gout arthritis), atransplant-related increase in cytokine production (particularly TNF, IL-1 and IL-6) mayactivate xanthine oxidase which, in turn, may be responsible for a further cytokine bout:deranged...
Book Chapter
Series: Contributions to Nephrology
Volume: 192
Published: 08 February 2018
10.1159/000484280
EISBN: 978-3-318-06251-9
... crystals deposition, what is the most appropriate UA target to be achieved, and how long does it need to be maintained and how does treatment need be modulated in patients with chronic kidney disease(CKD). Answers to such questions have been tentatively provided for patients with gout: in fact, the EULAR...